Way to go Dr. Payne. Keep up the good work! Published in "Science Direct" The full paper may be purchased on the Science Direct website.
Experimental regimen targeting the ependyma slows disease progression in four patients with amyotrophic lateral sclerosis
References and further reading may be available for this article. To view references and further reading you must purchase this article.
Anthony G. PayneCorresponding Author Contact Information, a, E-mail The Corresponding Author, E-mail The Corresponding Author
aWeller Health Institute and Laboratory, 3103 South El Camino Real, San Clemente, CA 92673, USA
Received 21 November 2008;
accepted 26 December 2008.
Available online 5 February 2009.
Summary
In this paper the author proposes that at least some forms of sporadic ALS (amyotrophic lateral sclerosis) arise due to the effects of neurotoxic compounds synthesized by defective ependymal cells in the brain. These cells produce cerebrospinal fluid (CSF) that is laden with neurotoxic compounds that bring about motor neuron die-off. Evidence is garnered from various animal studies to demonstrate the toxicity of CSF taken from ALS patients and by virtue of the proposed mechanism (defective ependymal cells). In addition, a regimen created by the author is introduced; a regimen that has been used by four (4) sporadic ALS patients since 2005 resulting in what appears to be a slowing of disease progression. All four patients have significantly outlived best estimates of their survival tendered by their neurologists.
http://www.healingcare4u.org/als-ependyma-hypothesis.html
Experimental regimen targeting the ependyma slows disease progression in four patients with amyotrophic lateral sclerosis
References and further reading may be available for this article. To view references and further reading you must purchase this article.
Anthony G. PayneCorresponding Author Contact Information, a, E-mail The Corresponding Author, E-mail The Corresponding Author
aWeller Health Institute and Laboratory, 3103 South El Camino Real, San Clemente, CA 92673, USA
Received 21 November 2008;
accepted 26 December 2008.
Available online 5 February 2009.
Summary
In this paper the author proposes that at least some forms of sporadic ALS (amyotrophic lateral sclerosis) arise due to the effects of neurotoxic compounds synthesized by defective ependymal cells in the brain. These cells produce cerebrospinal fluid (CSF) that is laden with neurotoxic compounds that bring about motor neuron die-off. Evidence is garnered from various animal studies to demonstrate the toxicity of CSF taken from ALS patients and by virtue of the proposed mechanism (defective ependymal cells). In addition, a regimen created by the author is introduced; a regimen that has been used by four (4) sporadic ALS patients since 2005 resulting in what appears to be a slowing of disease progression. All four patients have significantly outlived best estimates of their survival tendered by their neurologists.
http://www.healingcare4u.org/als-ependyma-hypothesis.html
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